Publikasjoner og forskningsresultater
Vitenskapelige publikasjoner
Sánchez-Pérez, Patricia; Mata, Ana; Torp, May-Kristin; López-Bernardo, Elia;
Heiestad, Christina Mathisen
; Aronsen, Jan Magnus; Molina-Iracheta, Antonio; Jiménez-Borreguero, Luis J.; García-Roves, Pablo; Costa, Ana S.H.; Frezza, Christian; Murphy, Michael P.; Stensløkken, Kåre-Olav; Cadenas, Susana
(2023).
Energy substrate metabolism, mitochondrial structure and oxidative stress after cardiac ischemia-reperfusion in mice lacking UCP3.
Free Radical Biology & Medicine.
Vol. 205.
https://doi.org/10.1016/j.freeradbiomed.2023.05.01
Melsom, Helene Støle;
Heiestad, Christina Mathisen
; Eftestøl, Einar; Torp, May-Kristin; Gundersen, Kristian; Bjørnebekk, Astrid; Thorsby, Per Medbøe; Stensløkken, Kåre-Olav; Hisdal, Jonny
(2022).
Reduced arterial elasticity after anabolic–androgenic steroid use in young adult males and mice.
Scientific Reports.
Vol. 12.
https://doi.org/10.1038/s41598-022-14065-5
Torp, May-Kristin; Ranheim, Trine; Schjalm, Camilla; Hjorth, Marit;
Heiestad, Christina Mathisen
; Dalen, Knut Tomas; Nilsson, Per; Mollnes, Tom Eirik; Pischke, Soeren; Lien, Egil; Vaage, Ingvar Jarle; Yndestad, Arne; Stensløkken, Kåre-Olav
(2022).
Intracellular Complement Component 3 Attenuated Ischemia-Reperfusion Injury in the Isolated Buffer-Perfused Mouse Heart and Is Associated With Improved Metabolic Homeostasis.
Frontiers in Immunology.
Vol. 13.
https://doi.org/10.3389/fimmu.2022.870811
Shah, Mohammed; He, Zhenhe; Rauf, Ali; Kalkhoran, Siavash Beikoghli;
Heiestad, Christina Mathisen
; Stensløkken, Kåre-Olav; Parish, Christopher R.; Soehnlein, Oliver; Arjun, Sapna; Davidson, Sean M.; Yellon, Derek
(2022).
Extracellular histones are a target in myocardial ischaemia-reperfusion injury.
Cardiovascular Research (CVR).
Vol. 118.
https://doi.org/10.1093/cvr/cvab139
Mariero, Lars Henrik; Torp, May-Kristin;
Heiestad, Christina Mathisen
; Baysa, Anton; Li, Yuchuan; Valen, Guro; Vaage, Ingvar Jarle; Stensløkken, Kåre-Olav
(2019).
Inhibiting nucleolin reduces inflammation induced by mitochondrial DNA in cardiomyocytes exposed to hypoxia and reoxygenation.
British Journal of Pharmacology.
Vol. 176.
https://doi.org/10.1111/bph.14830
